Alexander A. C. Rijsberman, Qualified Naturopath, Baar, Switzerland
Multi-resistance is one particular form of antibiotic resistance. We are talking about micro-organisms that have in effect become resistant to almost all antibiotics and virostatic agents. These are known as MRP or Multi-Resistant Pathogens.
In May 2014, the WHO (World Health Organization) published a wake-up call in a medical journal. The headline read: WHO raises the alarm. Antibiotic resistance is present everywhere. Wake-up call: even minor injuries that have been treated effectively for decades may now prove fatal.
According to Dr. Keiji Fukuda, Assistant Director-General — Health Security at the WHO in Geneva, the threat is imminent and affects people of all ages and in all countries. In the article, Dr. Fukuda calls for more intensive research in the field of antibiotic resistance. Time is short because third-generation antibiotics are failing to treat gonorrhoea, for instance. Worldwide, there are one million new cases of people contracting gonorrhoea each day.
This can be attributed to various causes:
– The frequent, often unnecessary use of antibiotics: antibiotics are prescribed for viral infections but serve no purpose in this respect.
– Forensic motivation: antibiotics are often prescribed in order to avoid being criticised for failure to prescribe antibiotics should subsequent complications arise.
– The use of antibiotics in the food industry. Antibiotics are often added to animal feed in order to increase yield.
Many of these antibiotics are related to those used in human medicine.
– Non-indicated use of antibiotics: powerful antibiotics are often prescribed for bacterial infections where penicillin, for example, would have sufficed.
– Unreliable treatment compliance on the part of patients. Hence pathogens are only partly destroyed and the surviving bacteria often increase their natural resistance.
– Natural survival of bacteria by genetic adaptation. Some of these adaptations can occur in just 20 minutes.
Excellent progress can be achieved with bioresonance in the areas of diagnosis and treatment. We have the option of using low deep frequencies to work on resistance. Regumed supply a small bottle of propolis with every new device. Propolis possesses anti-inflammatory properties and has generated good results in bacterial infections when used in conjunction with our method. Following a request from Mr. Brugemann, I have increasingly used Propolis and have incorporated it in a small series of tests. I should be delighted to show you my results later.
First, though, it is important to establish how bacteria develop resistance to antibiotics. This occurs in one of two ways: firstly, pathogens that are genetically resistant to the active substance are able to survive. Secondly, bacteria can alter their behaviour and very quickly adapt to changed conditions, thereby fooling non- genetic antibiotics.
This is known as persistence. Although genetic antibiotic resistance has been widely investigated, little is known about persistence.
The human immune system generally has two tasks. It must be able to respond effectively to external viruses and bacteria, and also prevent reactions against endogenous cells. Any form of inflammation results in dead cells. These dead cells represent a constant source of inflammation and are eliminated as endogenous debris by so-called sentinel cells (macrophages and dentritic cells). At the same time, the sentinel cells must recognise viruses and bacteria, and should trigger a targeted immune reaction in the form of inflammation with the help of T-cells in the lymph nodes. These sentinel cells must tread a fine line between, triggering a desirable inflammatory reaction against attacking pathogens and preventing inflammation from harming endogenous cells.
During a bacterial infection, sentinel cells absorb a small quantity of the pathogen. These sentinel cells then trigger an immune response. However, the system that basically helps the body to fight the pathogen is also at a disadvantage. Within the sentinel cell, part of the trapped bacteria stops multiplying rapidly. Antibiotics are virtually ineffective against bacteria that proliferate only slowly.
Research scientists based at the Swiss Federal Institute of Technology in Zurich infected mice with Salmonella and treated them with an antibiotic (Ciprofloxacin). They isolated sentinel cells from intestinal lymph nodes and detected surviving Salmonella. Scientists carried out tests to determine the ability of the bacteria to proliferate and the response of the latter to the antibiotic before comparing these with Salmonella taken from the animals’ intestines. Their findings were the following: in the Salmonella taken from the sentinel cells and intestines, there was evidence both of fast-growing bacteria that were killed by the antibiotic and also slowly proliferating bacteria against which the antibiotics proved ineffective. In the lymph node, however, the number of slowly proliferating, antibiotic-resistant bacteria sentinel cells had increased significantly.
This cannot be explained by sentinel cells preferring to absorb slowly proliferating bacteria but rather that the Salmonella inside the sentinel cells have altered their propagation behaviour in response to the prevailing nutrient-depleted conditions.
Scientists at the Institut for Theoretische Biologie (Institute of Theoretical Biology) have shown that the proliferation rate of bacteria in lymph nodes is dramatically reduced during the administration of antibiotics. This can be attributed to the fad that the rapidly dividing bacteria are killed by the antibiotic, resulting in an accumulation of slow-growing bacteria in the lymph nodes. During the course of an infection, persistent bacteria are quickly reconverted into fast-growing pathogens. An infection can therefore flare up again once the antibiotic is discontinued. This also explains why antibiotics should generally be taken for several days despite the fad that they actually take effect within minutes/hours.
To summarise or put it simply, bacteria have two modes of behaviour, namely “rapidly metabolising” or “slowly metabolising”.
This fact is also important for us in bioresonance therapy.
Researchers and the pharmaceutical industry are endeavouring to “crack” these bacteria in various ways.
The action is triggered at different points: in the cell walls, cell membranes, during folic acid metabolism, in ribosomes, during protein synthesis and DNA replication. As far as bioresonance is concerned, this is of little immediate interest but nice to know nonetheless. We have other options available to us.
I will now list a few multi-resistant bacteria that I frequently treat with bioresonance.
Escherichia coil and Klebsiella pneumoniae with NDM-1 strains (New Delhi metallo-13-lactamase 1)
This gene has emerged in Escherichia coil and Klebsiella pneumoniae, and is particularly prevalent in India and Pakistan. However, cases have also been reported in the UK, the Netherlands, Australia and Sweden. Infection often occurs during operations and cosmetic surgery. I would need one to two bioresonance sessions to treat this.
These bacteria have been reported since 1963, featuring a mutation in their penicillin-binding protein. They are now found worldwide and are increasingly posing a problem, especially in intensive care medicine. This strain can also appear as a colonising pathogen in the nasal and pharyngeal mucosa without the patient developing any symptoms. This leads to germ reservoirs that could infect other immunocompromised patients. Pathogen colonisation is particularly dangerous for doctors and hospital staff. I often detect this Staphylococcus in patients following surgery and dental procedures. Staphylococci and Streptococci are both stubborn and often require three to four treatments.
The bacterium is a hospital pathogen that displays multiple resistance to antibiotics through its metabolism and cell membrane structure. These are among the most common hospital pathogens. I often detect Pseudomonas aeruginosa in patients after they have been in hospital or undergone surgery. This hospital pathogen is greatly feared. It seldom proves resistant and is mostly eliminated with one to two sessions.
Salmonella D + TP + Typhimurium
Cause diarrhoea. I frequently detect Salmonella following the consumption of tainted foods such as poultry, eggs and mayonnaise, etc. It does not pose any problems for bioresonance.
I often detect this pathogen in children with persistent coughs. The barking cough usually disappears with two to three treatments.
An extremely stubborn pathogen. It takes 17 months to eradicate this pathogen. Particular attention should be paid to Borrelia because it can suddenly appear “out of the blue”. Only after a 17-month treatment period can I be certain that the pathogen has been fully eradicated. Borrelia infections also play a key role in a number of auto-immune diseases.
Campylobacter pylori + coli
These are often present in gastric/intestinal infections. They offer no resistance to treatment.
Proteus + Proteus reffgeri
These often appear as putrefactive bacteria in fruit and vegetables. These too can be easily treated.
The general trend I see is for nature to fight back. Bacteria and viruses become resistant to antibiotics and plant cells build up resistance to pesticide sprays. This highlights the complexity of the problem and how drastic things can become when resistance develops. At the present time certain bacterial infections are already being treated using cytostatic drugs, in other words with chemotherapy!
How can we treat these pathogens?
Let me summarise once again, because it is very important not to lose the overview. Fundamentally, the body has two types of cells:
– We have “rapidly metabolising” cells and
– very “slowly metabolising” cells.
So, when bacteria are detected, a certain proportion will enter the rapid cells and another proportion will invade the slow cells where they become persistent (“underlying”). Slow cells include nerves, muscles, the kidneys and the entire immune system. Note: this should not be confused with function as these cells operate very quickly in that respect. I am only talking here in terms of metabolism.
The Good Lord gave us deep sleep to regenerate these cells. At 8 Hz, we are resting, at 4 Hz we are asleep. We should then get down to 1 Hz three to four times during the night in order to achieve deep sleep (REM phase). My experience is that hardly any patients get beyond 2.3 Hz and so fail to reach 1 Hz.
As a result, bacteria (also nanoparticles, viruses and solvents) can directly penetrate our nerves and also infect the immune cells, in particular the sentinel cells and dentritic cells on the surface of the skin and mucosa.
These skin barriers no longer function properly and the underlying mast cells are unable to compensate for this lost barrier function. Hence, histamine is released and inflammation develops or too much histamine is released, falsely triggering allergies.
With regard to the slowly metabolising cells, I refer here to the causal level while for the rapidly metabolising cells I talk in terms of the symptom level. At symptom level, patients experience clear signs of discomfort and are able to confirm our diagnosis. At causal level, I have to explain things to the patient because he/she will not feel anything specific apart from perhaps feeling constantly tired, unable to shake off flu or only recovering very slowly. The patient will have a vague sense of being “under the weather”.
For testing purposes, I use the bacteria from the identification kit according to Dr. Schumacher — I have always found these to be the best option. I set the bioresonance device to program 3074.0 (regulate nerve function) and reprogram H+Di to Ai.
First of all, I look for the encapsulating components and place the appropriate material in the input cup (e.g. egg membrane, lactic acid, HCG, tumour capsule). Then I systematically search for bacteria. To do this, I examine the test set from left to right and once again from right to left. This means that I test the bacteria twice. The bacteria often hide and do not anticipate a second test. Using this trick, I mostly detect two or three different bacteria that were concealed during the first test. The bacteria are intertwined and in contact with each other. They are really clever and we have to outwit their survival strategy.
I treat the tested bacteria along with the encapsulating material using the same program, 3074.0 Ai, and all subsequent bacteria also with program 191.0. I begin by consciously treating in the low deep frequency range. I use the 2.3 Hz frequency to counteract the “slowly metabolising” bacteria in our slow cells, ie those that have proven to be persistent.
The slow bacteria may well flee and conceal themselves in fast tissue cells. To prevent this, I continue to treat with a fast program such as 191.0 — otherwise previous infections may recur. It is not unusual for bacteria to behave in this way, for they can adapt very quickly.
Tip: treatment times of eight to ten minutes are normal. Time and amplification should be adjusted accordingly.
To conclude, I would like to come back to the story of propolis. Mr. Brugemann asked me to investigate the supporting role of propolis. Propolis can be found in the small brown bottle supplied with every new device. To be honest, as far as I was concerned, it was nice to have the propolis but I was unable to test it in a wider context.
As I am always adopting different approaches, I also considered testing various antibiotics directly against the bacteria in addition to propolis, i.e. not in patients but directly against the ampoules of bacteria. In so doing I made a very interesting discovery, which I would like to share with you now.
The photo shows the test set-up with BICOM 2000. Tensor testing between the hand plates is suitable for this purpose.
The bacterial information is placed on the left hand plate and the medication information on the right hand plate, using the BICOM® device.
When I oscillated the various antibiotics directly against the bacteria, I had to set higher amplifications in order to achieve resonance (see table overleaf). This means that antibiotics only have a weak effect on bacteria or, in other words, very high doses are required, but I wondered whether the antibiotic was simply covering everything.
The amplifications were in some cases even higher than Ai 64 (see Borrelia). For the sake of clarity, these tests only concern effects in persistence. For fun, I also included various aromatic oils in the test. The example in the table refers to tea tree oil. Results averaging 1.30 in Ai were recorded with weak amplifications.
Regardless of the aromatic essences used, they all had more or less the same effect as each other, but were far more effective than the antibiotic.
And now for the surprise. I could not find any amplification with propolis. On the contrary, I had to work with dilutions in the potentiation program and, to my astonishment, at pretty high potencies. Bordetella pertussis is a typical example with D200!
And one further surprise. I also included the blue ampoule “Antibiotica” from Test Kit “Vaccines, …” in the test. Amazingly, this generated almost the same result as propolis. With this blue antibiotic ampoule, not only is it possible to eliminate antibiotics but it can also be used as an antibiotic support. Since then, I always include propolis and the blue antibiotic ampoule in the 2nd channel when treating bacterial infections.
Last but not least — have fun and good luck testing and treating your patients.
The World Health Organization (WHO) raises the alarm: antibiotic resistance is present everywhere
Wake-up call: even minor injuries may be fatal I warning to doctors
The World Health Organization has called for coordinated efforts to combat antimicrobial resistance, which is becoming increasingly prevalent worldwide.
By HELMUT LASCHET
GENEVA: In its first “Global Report on Antibiotic Resistance” and in response to the serious threat posed to public health across the globe, the World Health Organization (WHO) has discussed the diminishing efficacy of medicinal products in combatting infectious diseases. “This is no longer a future threat. It is imminent and can affect anyone, regardless of age and regardless of country,” pointed out Dr. Keiji Fukuda. Assistant Director-General for Health Security, when the report was presented in Geneva, on Wednesday.
“Without the urgent, coordinated action of numerous stakeholders, the world is facing a post-antibiotic era in which infections and minor injuries that have been treated effectively for decades may now prove fatal,” explained Dr. Fukuda.
Key points raised in the report:
- Antibiotics to combat carbapenem-resistant Klebsiella pneumoniae (CRKP) are now proving ineffective in more than 50% of patients in several countries. People suffering from pneumonia and sepsis as well as newborn infants and ICU (Intensive Care Unit) patients are particularly at risk.
- Quinolones, which were developed during the 1980s, have also proved ineffective in over 50% of patients treated with urinary tract infections, in many parts of the world.
“The threat is imminent and can affect anyone, regardless of age and regardless of country”
Dr. Keiji Fukuda
Assistant Director-General for Health Security, WHO, Geneva
Third-generation cephalosporins have proved ineffective in the treatment of gonorrhoea in Austria, Australia, Canada, France, Japan, Norway, South Africa, Sweden and the UK. One million people worldwide contract gonorrhoea on a daily basis.
→ The mortality rate from MRSA (methicillin-resistant Staphylococcus aureus) has increased by 64 percent.
The WHO is calling for improved hygiene and infection control in healthcare establishments and for vaccinations. The development of new diagnostic techniques and antibiotics is vital. Doctors should only prescribe antibiotics in cases where this is absolutely essential.
The WHO is appealing to political decision-makers and industry to intensify the research and development of new diagnostic tools and antibiotics. This should also apply to infection-related information policies.